Allergens and respiratory pollutants: The role of innate by Marc A. Williams PDF

By Marc A. Williams

ISBN-10: 1907568549

ISBN-13: 9781907568541

ISBN-10: 1908818069

ISBN-13: 9781908818065

Pt. 1. floor amendment thoughts -- pt. 2. Analytical suggestions and purposes

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Since exacerbations of allergic asthma are often driven by airway viral and/or bacterial infections, our studies (3) suggest that one link between environmental PM exposure and the exacerbations of pulmonary allergic conditions is a relative state of immune suppression and predisposition to infections as a consequence of down-regulated TLR2 and TLR4 cell surface expression. One could also postulate that the diminution in TLR expression in response to PM could represent an inherent mechanism to dampen the potential of pro-inflammatory airway hyperactivity and cellular damage.

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A number of candidate genes have been identified as possessing a contributory role in the development of allergic diseases. Foremost among these are those genes located in the chomosomal region 5q31–33, including IL-3, IL-4, IL-5, IL-9, IL-13, TGFβ and GM-CSF, that are collectively thought to program immune responsiveness from Th1 to Th2, isotytpe switching to IgE (as opposed to IgG) and activation of mast cells, basophils and eosinophils that contribute to many of the hallmarks of the immediate hypersensitivity response in allergic or atopic inflammation.

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Allergens and respiratory pollutants: The role of innate immunity by Marc A. Williams

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