By Marc A. Williams
Pt. 1. floor amendment thoughts -- pt. 2. Analytical suggestions and purposes
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Additional resources for Allergens and respiratory pollutants: The role of innate immunity
Since exacerbations of allergic asthma are often driven by airway viral and/or bacterial infections, our studies (3) suggest that one link between environmental PM exposure and the exacerbations of pulmonary allergic conditions is a relative state of immune suppression and predisposition to infections as a consequence of down-regulated TLR2 and TLR4 cell surface expression. One could also postulate that the diminution in TLR expression in response to PM could represent an inherent mechanism to dampen the potential of pro-inflammatory airway hyperactivity and cellular damage.
1992). The birth of immunology. III. The fate of the phagocytosis theory.
A number of candidate genes have been identified as possessing a contributory role in the development of allergic diseases. Foremost among these are those genes located in the chomosomal region 5q31–33, including IL-3, IL-4, IL-5, IL-9, IL-13, TGFβ and GM-CSF, that are collectively thought to program immune responsiveness from Th1 to Th2, isotytpe switching to IgE (as opposed to IgG) and activation of mast cells, basophils and eosinophils that contribute to many of the hallmarks of the immediate hypersensitivity response in allergic or atopic inflammation.
Allergens and respiratory pollutants: The role of innate immunity by Marc A. Williams